If our findings can be applied to humans, they should give us important insights into how type 2 diabetes may be prevented and treated.

Our findings suggest that the current human epidemic in type-2 diabetes may be a result of GnT-4a enzyme deficiency.

If you could somehow stimulate production of this enzyme, you might be able to render animals, and perhaps humans, resistant to high-fat diet-induced diabetes.

It may be that suppressing insulin production to some degree could be beneficial in such disorders.

This finding suggests new approaches to the prevention and treatment of diabetes.

It's always a big relief. Everyone expects us to get to state, but to actually be heading there now is really exciting.